High phosphate feeding promotes mineral and bone abnormalities in mice with chronic kidney disease | Zendy

Wei Ling Lau | Zendy; Michael Linnes | Zendy; Emily Y. Chu | Zendy; Brian L. Foster | Zendy; Bryan Bartley | Zendy; Martha J. Somerman | Zendy; Cecilia M. Giachelli | Zendy

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High phosphate feeding promotes mineral and bone abnormalities in mice with chronic kidney disease

Author(s) -

Wei Ling Lau,

Michael Linnes,

Emily Y. Chu,

Brian L. Foster,

Bryan Bartley,

Martha J. Somerman,

Cecilia M. Giachelli

Publication year - 2012

Publication title -

nephrology dialysis transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.654

H-Index - 168

eISSN - 1460-2385

pISSN - 0931-0509

DOI - 10.1093/ndt/gfs333

Subject(s) - medicine , endocrinology , kidney disease , calcification , ectopic calcification , renal osteodystrophy , cortical bone , bone remodeling , parathyroid hormone , hyperphosphatemia , chronic kidney disease mineral and bone disorder , alkaline phosphatase , kidney , bone disease , osteoporosis , calcium , pathology , chemistry , biochemistry , enzyme

Chronic kidney disease-mineral bone disorder (CKD-MBD) is a systemic syndrome characterized by imbalances in mineral homeostasis, renal osteodystrophy (ROD) and ectopic calcification. The mechanisms underlying this syndrome in individuals with chronic kidney disease (CKD) are not yet clear.

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