Copeptin, a surrogate marker for vasopressin, is associated with kidney function decline in subjects with autosomal dominant polycystic kidney disease
Author(s) -
Wendy E. Boertien,
Esther Meijer,
Debbie Zittema,
Marjan A. van Dijk,
Ton J. Rabelink,
M.H. Breuning,
J. Struck,
Stephan J. L. Bakker,
Dorien J.M. Peters,
Paul E. de Jong,
Ron T. Gansevoort
Publication year - 2012
Publication title -
nephrology dialysis transplantation
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.654
H-Index - 168
eISSN - 1460-2385
pISSN - 0931-0509
DOI - 10.1093/ndt/gfs070
Subject(s) - copeptin , medicine , vasopressin , surrogate endpoint , endocrinology , autosomal dominant polycystic kidney disease , renal function , kidney disease , polycystic kidney disease , kidney
Experimental studies have suggested that vasopressin plays a detrimental role in autosomal dominant polycystic kidney disease (ADPKD). It is, however, unknown whether endogenous vasopressin concentration is associated with kidney function decline in subjects with ADPKD.
Accelerating Research
Robert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom
Address
John Eccles HouseRobert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom