ADAM17 up-regulation in renal transplant dysfunction and non-transplant-related renal fibrosis | Zendy

Gemma M. Mulder | Zendy; Wynand B.W.H. Melenhorst | Zendy; Johanna W.A.M. Celie | Zendy; Niels J. Kloosterhuis | Zendy; JanLuuk Hillebrands | Zendy; R J Ploeg | Zendy; Marc A. Seelen | Zendy; Linda Visser | Zendy; Marcory C. R. F. van Dijk | Zendy; Harry van Goor | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

ADAM17 up-regulation in renal transplant dysfunction and non-transplant-related renal fibrosis

Author(s) -

Gemma M. Mulder,

Wynand B.W.H. Melenhorst,

Johanna W.A.M. Celie,

Niels J. Kloosterhuis,

JanLuuk Hillebrands,

R J Ploeg,

Marc A. Seelen,

Linda Visser,

Marcory C. R. F. van Dijk,

Harry van Goor

Publication year - 2011

Publication title -

nephrology dialysis transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.654

H-Index - 168

eISSN - 1460-2385

pISSN - 0931-0509

DOI - 10.1093/ndt/gfr583

Subject(s) - peritubular capillaries , mesangium , kidney , medicine , endocrinology , fibrosis , interstitial cell , renal function , epidermal growth factor , ctgf , growth factor , glomerulonephritis , receptor

Interstitial fibrosis and tubular atrophy (IF/TA) is an important cause of renal function loss and ischaemia-reperfusion (I/R) injury is considered to play an important role in its pathophysiology. The aim of the present study was to investigate the role of a disintegrin and metalloproteinase 17 (ADAM17) in human renal allograft disease and in experimental I/R injury of the kidney.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research