Lyme disease-associated glomerulonephritis | Zendy

Finnian R. Mc Causland | Zendy; Sophie Niedermaier | Zendy; V. Bijol | Zendy; Helmut G. Rennke | Zendy; Mary E. Choi | Zendy; John P. Forman | Zendy

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Lyme disease-associated glomerulonephritis

Author(s) -

Finnian R. Mc Causland,

Sophie Niedermaier,

V. Bijol,

Helmut G. Rennke,

Mary E. Choi,

John P. Forman

Publication year - 2011

Publication title -

nephrology dialysis transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.654

H-Index - 168

eISSN - 1460-2385

pISSN - 0931-0509

DOI - 10.1093/ndt/gfr335

Subject(s) - medicine , lyme disease , rash , glomerulonephritis , proteinuria , renal biopsy , membranoproliferative glomerulonephritis , nephropathy , mesangial proliferative glomerulonephritis , immunology , rapidly progressive glomerulonephritis , biopsy , pathology , dermatology , kidney , endocrinology , diabetes mellitus

We report two cases of Lyme disease-associated glomerulonephritis. A 57-year-old female presented with rash, volume overload, hypertension and rapidly progressive glomerulonephritis. Biopsy confirmed an immune complex-mediated, membranoproliferative lesion. She was treated successfully with steroids and antibiotics. In a second case, a 40-year-old male, with a previously known microscopic hematuria, presented with rash, arthralgias, new proteinuria and gross hematuria following a tick bite. Biopsy revealed focal proliferative IgA nephropathy. Treatment with steroids and antibiotics resulted in rapid resolution of findings. Acute Lyme disease may contribute to the development of de novo, or activation of previously quiescent, immune-mediated glomerular disease.

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