Depletion of kidney CD11c+ F4/80+ cells impairs the recovery process in ischaemia/reperfusion-induced acute kidney injury | Zendy

M.-G. Kim | Zendy; Chang Su Boo | Zendy; Y. S. Ko | Zendy; H. Y. Lee | Zendy; Won Yong Cho | Zendy; Hyoung Kyu Kim | Zendy; SangKyung Jo | Zendy

Open Access

Depletion of kidney CD11c+ F4/80+ cells impairs the recovery process in ischaemia/reperfusion-induced acute kidney injury

Author(s) -

M.-G. Kim,

Chang Su Boo,

Y. S. Ko,

H. Y. Lee,

Won Yong Cho,

Hyoung Kyu Kim,

SangKyung Jo

Publication year - 2010

Publication title -

nephrology dialysis transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.654

H-Index - 168

eISSN - 1460-2385

pISSN - 0931-0509

DOI - 10.1093/ndt/gfq183

Subject(s) - medicine , acute kidney injury , cd11c , kidney , ischemia , reperfusion injury , kidney disease , phenotype , gene , chemistry , biochemistry

Recent studies provided evidence of the potential role of CD11c(+) F4/80(+) dendritic subset in mediating injury and repair. The purpose of this study was to examine the role of kidney CD11c(+) F4/80(+) dendritic subset in the recovery phase of ischaemia/reperfusion injury (IRI).

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