Active vitamin D and its analogue, 22-oxacalcitriol, ameliorate puromycin aminonucleoside-induced nephrosis in rats | Zendy

Isao Matsui | Zendy; Takayuki Hamano | Zendy; K. Tomida | Zendy; Kazunori Inoue | Zendy; Yoshitsugu Takabatake | Zendy; Yasuyuki Nagasawa | Zendy; Noritaka Kawada | Zendy; Takahito Ito | Zendy; H. Kawachi | Zendy; Hiromi Rakugi | Zendy; Enyu Imai | Zendy; Yoshitaka Isaka | Zendy

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Active vitamin D and its analogue, 22-oxacalcitriol, ameliorate puromycin aminonucleoside-induced nephrosis in rats

Author(s) -

Isao Matsui,

Takayuki Hamano,

K. Tomida,

Kazunori Inoue,

Yoshitsugu Takabatake,

Yasuyuki Nagasawa,

Noritaka Kawada,

Takahito Ito,

H. Kawachi,

Hiromi Rakugi,

Enyu Imai,

Yoshitaka Isaka

Publication year - 2009

Publication title -

nephrology dialysis transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.654

H-Index - 168

eISSN - 1460-2385

pISSN - 0931-0509

DOI - 10.1093/ndt/gfp117

Subject(s) - podocyte , nephrin , podocin , endocrinology , nephrosis , medicine , calcitriol , calcitriol receptor , vitamin d and neurology , vitamin d deficiency , proteinuria , kidney

Recent studies have demonstrated that podocyte injury, which results in proteinuria, leads to tubulointerstitial fibrosis. Although some studies have revealed that vitamin D administration protects renal structure and function in mesangial cell proliferative and/or excessive matrix productive models, the effects of vitamin D on podocyte injury have remained uncertain.

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