Familial ATTR amyloidosis: microalbuminuria as a predictor of symptomatic disease and clinical nephropathy | Zendy

Luísa Lobato | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Familial ATTR amyloidosis: microalbuminuria as a predictor of symptomatic disease and clinical nephropathy

Author(s) -

Luísa Lobato

Publication year - 2003

Publication title -

nephrology dialysis transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.654

H-Index - 168

eISSN - 1460-2385

pISSN - 0931-0509

DOI - 10.1093/ndt/18.3.532

Subject(s) - medicine , amyloidosis , asymptomatic , microalbuminuria , nephropathy , transthyretin , proteinuria , gastroenterology , kidney disease , renal function , endocrinology , kidney , diabetes mellitus

Portuguese type familial amyloid polyneuropathy (FAP) is a neuropathic amyloidosis caused by a mutant transthyretin (TTR). Varying degrees of renal involvement have been reported. Our aim was to assess the value of microalbuminuria (MA) for predicting clinical neurological disease and overt nephropathy in TTR-related amyloidosis.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research