Does circulating erythropoietin reflect progression of IgA nephropathy? Comparison with urinary N-acetyl- -D-glucosaminidase | Zendy

Toshihiko Machiguchi | Zendy; Hiroshi Yoshida | Zendy; Satomi Yonemoto | Zendy; Tamotsu Minakata | Zendy; K. Nomura | Zendy; Eri Muso | Zendy; T. Tamura | Zendy; S Sasayama | Zendy

Open Access

Does circulating erythropoietin reflect progression of IgA nephropathy? Comparison with urinary N-acetyl- -D-glucosaminidase

Author(s) -

Toshihiko Machiguchi,

Hiroshi Yoshida,

Satomi Yonemoto,

Tamotsu Minakata,

K. Nomura,

Eri Muso,

T. Tamura,

S Sasayama

Publication year - 1999

Publication title -

nephrology dialysis transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.654

H-Index - 168

eISSN - 1460-2385

pISSN - 0931-0509

DOI - 10.1093/ndt/14.3.635

Subject(s) - medicine , erythropoietin , creatinine , nephropathy , endocrinology , renal function , urinary system , proteinuria , renal biopsy , glomerulosclerosis , radioimmunoassay , glomerulonephritis , biopsy , kidney , urology , diabetes mellitus

Recent reports describe that erythropoietin (Epo) is produced by peritubular interstitial fibroblast-like cells in response to a hypoxic stimulus. We studied serum Epo levels as a possible marker of tubulointerstitial damage in the progression of IgA nephropathy (IgAN), in comparison with urinary (u-) levels of N-acetyl-beta-D-glucosaminidase (NAG), which is mainly derived from proximal tubular cells and is used as a marker of tubular damage.

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