Regulation of Mitochondrial Oxidative Metabolism by Tumor Suppressor FLCN | Zendy

Hisashi Hasumi | Zendy; Masaya Baba | Zendy; Yukiko Hasumi | Zendy; Ying Huang | Zendy; HyoungBin Oh | Zendy; Robert M. Hughes | Zendy; Mara E. Klein | Zendy; Shoichi Takikita | Zendy; Kunio Nagashima | Zendy; Laura S. Schmidt | Zendy; W. Marston Linehan | Zendy

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Regulation of Mitochondrial Oxidative Metabolism by Tumor Suppressor FLCN

Author(s) -

Hisashi Hasumi,

Masaya Baba,

Yukiko Hasumi,

Ying Huang,

HyoungBin Oh,

Robert M. Hughes,

Mara E. Klein,

Shoichi Takikita,

Kunio Nagashima,

Laura S. Schmidt,

W. Marston Linehan

Publication year - 2012

Publication title -

jnci journal of the national cancer institute

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 5.797

H-Index - 356

eISSN - 1460-2105

pISSN - 0027-8874

DOI - 10.1093/jnci/djs418

Subject(s) - folliculin , oxidative metabolism , oxidative phosphorylation , mitochondrion , suppressor , metabolism , microbiology and biotechnology , biology , chemistry , cancer research , biochemistry , cancer , genetics , gene

Birt-Hogg-Dubé (BHD) syndrome is a hereditary hamartoma syndrome that predisposes patients to develop hair follicle tumors, lung cysts, and kidney cancer. Genetic studies of BHD patients have uncovered the causative gene, FLCN, but its function is incompletely understood.

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