Phylogenetic background and carriage of pathogenicity island-like domains in relation to antibiotic resistance profiles among Escherichia coli urosepsis isolates | Zendy

Véronique Houdouin | Zendy; Stéphane Bonacorsi | Zendy; Philippe Bidet | Zendy; Martine Bingen-Bidois | Zendy; Dominique Barraud | Zendy; Édouard Bingen | Zendy

Open Access

Phylogenetic background and carriage of pathogenicity island-like domains in relation to antibiotic resistance profiles among Escherichia coli urosepsis isolates

Author(s) -

Véronique Houdouin,

Stéphane Bonacorsi,

Philippe Bidet,

Martine Bingen-Bidois,

Dominique Barraud,

Édouard Bingen

Publication year - 2006

Publication title -

journal of antimicrobial chemotherapy

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.124

H-Index - 194

eISSN - 1460-2091

pISSN - 0305-7453

DOI - 10.1093/jac/dkl326

Subject(s) - nalidixic acid , biology , virulence , ampicillin , carriage , escherichia coli , microbiology and biotechnology , pathogenicity , locus (genetics) , antibiotic resistance , phylogenetic tree , genetics , sulfamethoxazole , antibiotics , pathogenicity island , linkage (software) , enterobacteriaceae , gene , medicine , pathology

We studied 100 well-characterized E. coli blood isolates from patients with urosepsis for their susceptibility to nalidixic acid, ampicillin and trimethoprim-sulfamethoxazole, according to prevalence of virulence factors, phylogenetic groups and subgroups, PAI II(J96)-like domains (determined by physical linkage of cnf1, hly and hra) and PAI I(CFT073)-like domains (determined by physical linkage of papGII to the hly locus). Nalidixic acid resistance was associated with a lower prevalence of sfa/foc, K1 antigen, pathogenicity island II(J96)-like domains, subgroup B2/I and a shift towards group A.

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