The Complement C3a Receptor Is Critical in Defense against Chlamydia psittaci in Mouse Lung Infection and Required for Antibody and Optimal T Cell Response | Zendy

Pavel Dutow | Zendy; Beate Fehlhaber | Zendy; Jenny Bode | Zendy; Robert Laudeley | Zendy; Claudia Rheinheimer | Zendy; Silke Glage | Zendy; Rick A. Wetsel | Zendy; Oliver Pabst | Zendy; Andreas Klos | Zendy

Open Access

The Complement C3a Receptor Is Critical in Defense against Chlamydia psittaci in Mouse Lung Infection and Required for Antibody and Optimal T Cell Response

Author(s) -

Pavel Dutow,

Beate Fehlhaber,

Jenny Bode,

Robert Laudeley,

Claudia Rheinheimer,

Silke Glage,

Rick A. Wetsel,

Oliver Pabst,

Andreas Klos

Publication year - 2013

Publication title -

the journal of infectious diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.69

H-Index - 252

eISSN - 1537-6613

pISSN - 0022-1899

DOI - 10.1093/infdis/jit640

Subject(s) - immunology , biology , chlamydia psittaci , antibody , immune system , anaphylatoxin , adoptive cell transfer , chlamydia , acquired immune system , complement system , cd8 , immunity , t cell

The complement system protects against extracellular pathogens and links innate and adaptive immunity. In this study, we investigated the anaphylatoxin C3a receptor (C3aR) in Chlamydia psittaci lung infection and elucidated C3a-dependent adaptive immune mechanisms.

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