Inheritance of the Lysozyme Inhibitor Ivy Was an Important Evolutionary Step by Yersinia pestis to Avoid the Host Innate Immune Response | Zendy

Anne Derbise | Zendy; François Pierre | Zendy; Maud Merchez | Zendy; Elizabeth Pradel | Zendy; Sabrina Laouami | Zendy; Isabelle Ricard | Zendy; JeanClaude Sirard | Zendy; Jill M. Fritz | Zendy; Nadine Lemaître | Zendy; Henry T. Akinbi | Zendy; Ivo G. Boneca | Zendy; Florent Sebbane | Zendy

Open Access

Inheritance of the Lysozyme Inhibitor Ivy Was an Important Evolutionary Step by Yersinia pestis to Avoid the Host Innate Immune Response

Author(s) -

Anne Derbise,

François Pierre,

Maud Merchez,

Elizabeth Pradel,

Sabrina Laouami,

Isabelle Ricard,

JeanClaude Sirard,

Jill M. Fritz,

Nadine Lemaître,

Henry T. Akinbi,

Ivo G. Boneca,

Florent Sebbane

Publication year - 2013

Publication title -

the journal of infectious diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.69

H-Index - 252

eISSN - 1537-6613

pISSN - 0022-1899

DOI - 10.1093/infdis/jit057

Subject(s) - lysozyme , virulence , biology , yersinia pestis , microbiology and biotechnology , pathogen , mutant , yersinia pseudotuberculosis , plague (disease) , immune system , yersinia , yersiniosis , gene , virology , bacteria , genetics , enterobacteriaceae , escherichia coli , history , archaeology

Yersinia pestis (the plague bacillus) and its ancestor, Yersinia pseudotuberculosis (which causes self-limited bowel disease), encode putative homologues of the periplasmic lysozyme inhibitor Ivy and the membrane-bound lysozyme inhibitor MliC. The involvement of both inhibitors in virulence remains subject to debate.

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