Cortical GABA in Subjects at Ultra-High Risk of Psychosis: Relationship to Negative Prodromal Symptoms | Zendy

Gemma Modinos | Zendy; Fatma Şimşek | Zendy; Jamie Horder | Zendy; Matthijs G. Bossong | Zendy; Ilaria Bonoldi | Zendy; Matilda Azis | Zendy; Jesús Pérez | Zendy; Matthew R. Broome | Zendy; David J. Lythgoe | Zendy; James Stone | Zendy; Oliver Howes | Zendy; Declan Murphy | Zendy; Anthony A. Grace | Zendy; Paul Allen | Zendy; Philip McGuire | Zendy

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Cortical GABA in Subjects at Ultra-High Risk of Psychosis: Relationship to Negative Prodromal Symptoms

Author(s) -

Gemma Modinos,

Fatma Şimşek,

Jamie Horder,

Matthijs G. Bossong,

Ilaria Bonoldi,

Matilda Azis,

Jesús Pérez,

Matthew R. Broome,

David J. Lythgoe,

James Stone,

Oliver Howes,

Declan Murphy,

Anthony A. Grace,

Paul Allen,

Philip McGuire

Publication year - 2017

Publication title -

the international journal of neuropsychopharmacology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.897

H-Index - 107

eISSN - 1469-5111

pISSN - 1461-1457

DOI - 10.1093/ijnp/pyx076

Subject(s) - psychosis , psychology , psychiatry , clinical psychology , neuroscience , medicine

Whilst robust preclinical and postmortem evidence suggests that altered GABAergic function is central to the development of psychosis, little is known about whether it is altered in subjects at ultra-high risk of psychosis, or its relationship to prodromal symptoms.

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