MicroRNA-143 Targets ATG2B to Inhibit Autophagy and Increase Inflammatory Responses in Crohn’s Disease | Zendy

Xutao Lin | Zendy; Xiaobin Zheng | Zendy; Dejun Fan | Zendy; Qiuqiong Yao | Zendy; Jiancong Hu | Zendy; Lei Lian | Zendy; Xiaojian Wu | Zendy; Ping Lan | Zendy; Xiaosheng He | Zendy

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MicroRNA-143 Targets ATG2B to Inhibit Autophagy and Increase Inflammatory Responses in Crohn’s Disease

Author(s) -

Xutao Lin,

Xiaobin Zheng,

Dejun Fan,

Qiuqiong Yao,

Jiancong Hu,

Lei Lian,

Xiaojian Wu,

Ping Lan,

Xiaosheng He

Publication year - 2018

Publication title -

inflammatory bowel diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.932

H-Index - 146

eISSN - 1536-4844

pISSN - 1078-0998

DOI - 10.1093/ibd/izx075

Subject(s) - autophagy , microrna , inflammatory bowel disease , atg16l1 , inflammation , untranslated region , luciferase , biology , proinflammatory cytokine , tumor necrosis factor alpha , reverse transcription polymerase chain reaction , microbiology and biotechnology , cancer research , messenger rna , immunology , medicine , pathology , apoptosis , gene , disease , transfection , genetics

Dysfunctional autophagy is recognized as a contributing factor in many chronic inflammatory diseases, including Crohn's disease (CD). Genetic analyses have found that microRNA (miRNA) levels are altered in the intestinal tissues of CD patients.

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