Open AccessPlatelet-derived TGF-β1 mediates the down-modulation of NKG2D expression and may be responsible for impaired natural killer (NK) cytotoxicity in women with endometriosis
Author(s) -
SunWei Guo,
Yanbo Du,
Xishi Liu
Publication year - 2016
Publication title -
human reproduction
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.446
H-Index - 226
eISSN - 1460-2350
pISSN - 0268-1161
DOI - 10.1093/humrep/dew057
Subject(s) - nkg2d , endometriosis , platelet , natural killer cell , platelet activation , peritoneal fluid , immunology , cytotoxic t cell , medicine , endocrinology , chemistry , in vitro , biochemistry
Does platelet-derived transforming growth factor-β1 (TGF-β1) have any role in the reduced cytotoxicity of natural killer (NK) cells in women with endometriosis?
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