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Overexpression of chloride channel-3 is associated with the increased migration and invasion ability of ectopic endometrial cells from patients with endometriosis
Author(s) -
Yutao Guan,
Yanqing Huang,
Jiabao Wu,
Zhiqin Deng,
Yuan Wang,
Zhou-yi Lai,
Haibo Wang,
Xiaoxue Sun,
Yali Zhu,
Miaomiao Du,
Linyan Zhu,
Lixin Chen,
Liwei Wang
Publication year - 2016
Publication title -
human reproduction
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.446
H-Index - 226
eISSN - 1460-2350
pISSN - 0268-1161
DOI - 10.1093/humrep/dew034
Subject(s) - endometriosis , gene knockdown , matrigel , gentamicin protection assay , stromal cell , ectopic expression , immunohistochemistry , small interfering rna , cancer research , pelvic pain , matrix metalloproteinase , medicine , cell migration , transfection , andrology , biology , cell , cell culture , pathology , metastasis , angiogenesis , surgery , cancer , genetics
Is chloride channel-3 (ClC-3) involved in regulating the biological behavior of endometrial stromal cells (ESCs)?

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