Control of extravillous trophoblast function by the eotaxins CCL11, CCL24 and CCL26 | Zendy

S. E. Chau | Zendy; Padma Murthi | Zendy; M H Wong | Zendy; Guy Whitley | Zendy; Shaun P. Brennecke | Zendy; Rosemary J. Keogh | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Control of extravillous trophoblast function by the eotaxins CCL11, CCL24 and CCL26

Author(s) -

S. E. Chau,

Padma Murthi,

M H Wong,

Guy Whitley,

Shaun P. Brennecke,

Rosemary J. Keogh

Publication year - 2013

Publication title -

human reproduction

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.446

H-Index - 226

eISSN - 1460-2350

pISSN - 0268-1161

DOI - 10.1093/humrep/det060

Subject(s) - trophoblast , ccl11 , chemokine , decidua , microbiology and biotechnology , placentation , cell migration , biology , extracellular matrix , cxc chemokine receptors , zymography , immunology , andrology , placenta , chemokine receptor , matrix metalloproteinase , cell culture , eotaxin , medicine , inflammation , pregnancy , biochemistry , genetics , fetus

What are the effects of the eotaxin group of chemokines (CCL11, CCL24 and CCL26) on extravillous trophoblast (EVT) functions important during uterine decidual vessel remodelling?

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research