The role of Foxp3+ regulatory T-cells in endometriosis: a potential controlling mechanism for a complex, chronic immunological condition | Zendy

Marina Berbic | Zendy; Alison J. HeyCunningham | Zendy; Cecilia Ng | Zendy; Natsuko Tokushige | Zendy; Subha P. Ganewatta | Zendy; Robert Markham | Zendy; Pamela J. Russell | Zendy; Ian S. Fraser | Zendy

Open Access

The role of Foxp3+ regulatory T-cells in endometriosis: a potential controlling mechanism for a complex, chronic immunological condition

Author(s) -

Marina Berbic,

Alison J. HeyCunningham,

Cecilia Ng,

Natsuko Tokushige,

Subha P. Ganewatta,

Robert Markham,

Pamela J. Russell,

Ian S. Fraser

Publication year - 2010

Publication title -

human reproduction

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.446

H-Index - 226

eISSN - 1460-2350

pISSN - 0268-1161

DOI - 10.1093/humrep/deq020

Subject(s) - foxp3 , endometriosis , mechanism (biology) , immunology , medicine , biology , immune system , gynecology , philosophy , epistemology

Endometriosis is an inflammatory condition, associated with highly dysregulated immune response at both uterine and peritoneal levels. Surprisingly, Foxp3+ regulatory T-cells, which control and suppress a range of immune responses, have not previously been investigated in endometriosis.

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