Molecular mechanisms of ovarian hyperstimulation syndrome: paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability | Zendy

Miriam Rodewald | Zendy; Daniel Herr | Zendy; W. Colin Duncan | Zendy; Hamish M. Fraser | Zendy; G Hack | Zendy; Р. Конрад | Zendy; F. Gagsteiger | Zendy; R. Kreienberg | Zendy; Christine Wulff | Zendy

Open Access

Molecular mechanisms of ovarian hyperstimulation syndrome: paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability

Author(s) -

Miriam Rodewald,

Daniel Herr,

W. Colin Duncan,

Hamish M. Fraser,

G Hack,

Р. Конрад,

F. Gagsteiger,

R. Kreienberg,

Christine Wulff

Publication year - 2009

Publication title -

human reproduction

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.446

H-Index - 226

eISSN - 1460-2350

pISSN - 0268-1161

DOI - 10.1093/humrep/den479

Subject(s) - ovarian hyperstimulation syndrome , claudin , in vitro , paracrine signalling , andrology , permeability (electromagnetism) , endocrinology , medicine , in vitro fertilisation , biology , chemistry , microbiology and biotechnology , tight junction , embryo , membrane , genetics , receptor

Ovarian hyperstimulation syndrome (OHSS) is a potentially life-threatening complication of ovarian stimulation associated with severe vascular hyperpermeability. Primary co-cultures of human luteinized granulosa cells (LGCs) and human umbilical vein endothelial cells (HUVECs) were used as a model of steroidgenic/endothelial cell interaction in OHSS.

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