Metoclopramide-induced hyperprolactinaemia caused marked decline in pinopodes and pregnancy rates in mice | Zendy

Michele Q. Panzan | Zendy; J. M. S. | Zendy; E.L.A. Motta | Zendy; Edna Haapalainen | Zendy; Manuel de Jesus Simões | Zendy; H. A. Baptista | Zendy; Mauro Abi Haidar | Zendy; E C Baracat | Zendy

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Metoclopramide-induced hyperprolactinaemia caused marked decline in pinopodes and pregnancy rates in mice

Author(s) -

Michele Q. Panzan,

J. M. S.,

E.L.A. Motta,

Edna Haapalainen,

Manuel de Jesus Simões,

H. A. Baptista,

Mauro Abi Haidar,

E C Baracat

Publication year - 2006

Publication title -

human reproduction

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.446

H-Index - 226

eISSN - 1460-2350

pISSN - 0268-1161

DOI - 10.1093/humrep/del093

Subject(s) - estrous cycle , hyperprolactinaemia , endocrinology , estrogen , endometrium , medicine , embryo transfer , embryo , prolactin , pregnancy , uterus , population , luteal phase , andrology , uterine horns , biology , hormone , genetics , environmental health , microbiology and biotechnology

The impact of hyperprolactinaemia on endometrial function, along with embryo implantation, has been the subject of discussion. This article examines whether experimental hyperprolactinaemia can affect mouse ovarian function, endometrial pinopodes and embryo implantation.

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