Omentin attenuates atherosclerotic lesion formation in apolipoprotein E-deficient mice | Zendy

Mizuho HiramatsuIto | Zendy; Rei Shibata | Zendy; Koji Ohashi | Zendy; Yusuke Uemura | Zendy; Noriyoshi Kanemura | Zendy; Takahiro Kambara | Zendy; Takashi Enomoto | Zendy; Daisuke Yuasa | Zendy; Kazuhiro Matsuo | Zendy; Masanori Ito | Zendy; Satoko Hayakawa | Zendy; Hayato Ogawa | Zendy; Naoya Otaka | Zendy; Shinji Kihara | Zendy; Toyoaki Murohara | Zendy; Noriyuki Ouchi | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Omentin attenuates atherosclerotic lesion formation in apolipoprotein E-deficient mice

Author(s) -

Mizuho HiramatsuIto,

Rei Shibata,

Koji Ohashi,

Yusuke Uemura,

Noriyoshi Kanemura,

Takahiro Kambara,

Takashi Enomoto,

Daisuke Yuasa,

Kazuhiro Matsuo,

Masanori Ito,

Satoko Hayakawa,

Hayato Ogawa,

Naoya Otaka,

Shinji Kihara,

Toyoaki Murohara,

Noriyuki Ouchi

Publication year - 2015

Publication title -

cardiovascular research

Language(s) - Uncategorized

Resource type - Journals

SCImago Journal Rank - 2.774

H-Index - 219

eISSN - 1755-3245

pISSN - 0008-6363

DOI - 10.1093/cvr/cvv282

Subject(s) - apolipoprotein b , lesion , apolipoprotein e , medicine , endocrinology , cardiology , pathology , cholesterol , disease

Obesity is associated with the development of atherosclerosis. We previously demonstrated that omentin is a circulating adipokine that is downregulated in association with atherosclerotic diseases. Here, we examined the impact of omentin on the development of atherosclerosis with gain-of-function genetic manipulations and dissected its potential mechanism.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research