Molecular basis of selective atrial fibrosis due to overexpression of transforming growth factor-β1 | Zendy

Dolkun Rahmutula | Zendy; Gregory M. Marcus | Zendy; Emily Wilson | Zendy; Chunhua Ding | Zendy; Yuanyuan Xiao | Zendy; Agnès Paquet | Zendy; Rebecca Barbeau | Zendy; Andrea J. Barczak | Zendy; David J. Erle | Zendy; Jeffrey E. Olgin | Zendy

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Molecular basis of selective atrial fibrosis due to overexpression of transforming growth factor-β1

Author(s) -

Dolkun Rahmutula,

Gregory M. Marcus,

Emily Wilson,

Chunhua Ding,

Yuanyuan Xiao,

Agnès Paquet,

Rebecca Barbeau,

Andrea J. Barczak,

David J. Erle,

Jeffrey E. Olgin

Publication year - 2013

Publication title -

cardiovascular research

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.774

H-Index - 219

eISSN - 1755-3245

pISSN - 0008-6363

DOI - 10.1093/cvr/cvt074

Subject(s) - fibrosis , medicine , transforming growth factor , atrial fibrillation , cardiac fibrosis , ventricle , myocardial fibrosis , endocrinology , receptor

Animal studies show that transforming growth factor-β1 (TGF-β1) is an important mediator of atrial fibrosis and atrial fibrillation (AF). This study investigated the role of TGF-β1 in human AF and the mechanism of atrial-selective fibrosis.

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