Inhibition of brain angiotensin III attenuates sympathetic hyperactivity and cardiac dysfunction in rats post-myocardial infarction | Zendy

Bing Huang | Zendy; Monir Ahmad | Zendy; Roselyn White | Zendy; Yannick Marc | Zendy; Catherine LlorensCortès | Zendy; Frans H. H. Leenen | Zendy

Open Access

Inhibition of brain angiotensin III attenuates sympathetic hyperactivity and cardiac dysfunction in rats post-myocardial infarction

Author(s) -

Bing Huang,

Monir Ahmad,

Roselyn White,

Yannick Marc,

Catherine LlorensCortès,

Frans H. H. Leenen

Publication year - 2012

Publication title -

cardiovascular research

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.774

H-Index - 219

eISSN - 1755-3245

pISSN - 0008-6363

DOI - 10.1093/cvr/cvs420

Subject(s) - medicine , myocardial infarction , cardiology , angiotensin ii , angiotensin 1 , cardiac dysfunction , heart failure , blood pressure

In rats post-myocardial infarction (MI), activation of angiotensinergic pathways in the brain contributes to sympathetic hyperactivity and progressive left ventricle (LV) dysfunction. The present study examined whether angiotensin III (Ang III) is one of the main effector peptides of the brain renin-angiotensin system controlling these effects.

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