Infarct-remodelled hearts with limited oxidative capacity boost fatty acid oxidation after conditioning against ischaemia/reperfusion injury
Author(s) -
PhingHow Lou,
Liyan Zhang,
Eliana Lucchinetti,
M. Heck,
Andreas Affolter,
Manoj Gandhi,
Petra C. Kienesberger,
Martin Hersberger,
Alexander S. Clanachan,
Michael Zaugg
Publication year - 2012
Publication title -
cardiovascular research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.774
H-Index - 219
eISSN - 1755-3245
pISSN - 0008-6363
DOI - 10.1093/cvr/cvs323
Subject(s) - ischemia , medicine , reperfusion injury , oxidative phosphorylation , cardiology , oxidative metabolism , metabolism , biology , biochemistry
Infarct-remodelled hearts are less amenable to protection against ischaemia/reperfusion. Understanding preservation of energy metabolism in diseased vs. healthy hearts may help to develop anti-ischaemic strategies effective also in jeopardized myocardium.
Accelerating Research
Robert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom
Address
John Eccles HouseRobert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom