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Infarct-remodelled hearts with limited oxidative capacity boost fatty acid oxidation after conditioning against ischaemia/reperfusion injury
Author(s) -
PhingHow Lou,
Liyan Zhang,
Eliana Lucchinetti,
M. Heck,
Andreas Affolter,
Manoj Gandhi,
Petra C. Kienesberger,
Martin Hersberger,
Alexander S. Clanachan,
Michael Zaugg
Publication year - 2012
Publication title -
cardiovascular research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.774
H-Index - 219
eISSN - 1755-3245
pISSN - 0008-6363
DOI - 10.1093/cvr/cvs323
Subject(s) - ischemia , medicine , reperfusion injury , oxidative phosphorylation , cardiology , oxidative metabolism , metabolism , biology , biochemistry
Infarct-remodelled hearts are less amenable to protection against ischaemia/reperfusion. Understanding preservation of energy metabolism in diseased vs. healthy hearts may help to develop anti-ischaemic strategies effective also in jeopardized myocardium.

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