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Relaxin improves TNF-α-induced endothelial dysfunction: the role of glucocorticoid receptor and phosphatidylinositol 3-kinase signalling
Author(s) -
Thomas Dschietzig,
Anna Brecht,
Cornelia Bartsch,
Gert Baumann,
Karl Stangl,
Konstantin Alexiou
Publication year - 2012
Publication title -
cardiovascular research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.774
H-Index - 219
eISSN - 1755-3245
pISSN - 0008-6363
DOI - 10.1093/cvr/cvs149
Subject(s) - phosphatidylinositol , glucocorticoid receptor , kinase , relaxin , endocrinology , medicine , signalling , receptor , signal transduction , glucocorticoid , endothelial dysfunction , microbiology and biotechnology , biology
Human relaxin-2 influences renal and cardiovascular functions. We investigated its effects on experimental endothelial dysfunction.

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