Role of nuclear unphosphorylated STAT3 in angiotensin II type 1 receptor-induced cardiac hypertrophy | Zendy

Hong Yue | Zendy; Wěi Li | Zendy; Russell Desnoyer | Zendy; Sadashiva S. Karnik | Zendy

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Role of nuclear unphosphorylated STAT3 in angiotensin II type 1 receptor-induced cardiac hypertrophy

Author(s) -

Hong Yue,

Wěi Li,

Russell Desnoyer,

Sadashiva S. Karnik

Publication year - 2009

Publication title -

cardiovascular research

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.774

H-Index - 219

eISSN - 1755-3245

pISSN - 0008-6363

DOI - 10.1093/cvr/cvp285

Subject(s) - angiotensin ii , stat3 , chromatin immunoprecipitation , gene knockdown , biology , muscle hypertrophy , transcription factor , medicine , microbiology and biotechnology , endocrinology , phosphorylation , pressure overload , gene expression , chemistry , gene , promoter , biochemistry , blood pressure , cardiac hypertrophy

Cardiac hypertrophy is a risk factor independent of blood pressure; however, the mechanisms that distinguish pathological remodelling due to local cues from pressure overload are unresolved. This study was aimed at discovering a novel gene expression mechanism in heart failure.

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