Reversibility of oxalate nephropathy in a kidney transplant recipient with prior gastric bypass surgery
Author(s) -
Christian Goul Sørensen,
Christian Lodberg Hvas,
I.M. Thomsen,
Bente Jespersen
Publication year - 2020
Publication title -
clinical kidney journal
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.033
H-Index - 40
eISSN - 2048-8513
pISSN - 2048-8505
DOI - 10.1093/ckj/sfaa254
Subject(s) - medicine , dialysis , nephropathy , oxalate , calcium oxalate , kidney , renal function , gastric bypass surgery , surgery , hemodialysis , transplantation , urology , kidney transplantation , kidney stones , peritoneal dialysis , gastroenterology , obesity , gastric bypass , diabetes mellitus , endocrinology , weight loss , calcium , chemistry , organic chemistry
Bariatric surgery is an acknowledged treatment for obesity and related comorbidities with beneficial effects on kidney function. However, bariatric surgery can also lead to secondary hyperoxaluria and oxalate nephropathy, resulting in end-stage kidney disease in both native and transplanted kidneys. We present a 66-year-old man who was in need of dialysis 3 months after kidney transplantation due to recurrent oxalate nephropathy. Intensified haemodialysis together with increased liquid intake, dietary restrictions of oxalate and fat and supplementation with calcium citrate and a bile acid binder were applied. Graft function improved and the patient did not require dialysis during the following 8 months.
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