Posttreatment Reactivation of Tuberculosis in Mice Caused byMycobacterium tuberculosisDisrupted inmce1R | Zendy

ChanIck Cheigh | Zendy; Ryan H. Senaratne | Zendy; Yujiro Uchida | Zendy; Nicola Casali | Zendy; Lon V. Kendall | Zendy; Lee W. Riley | Zendy

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Posttreatment Reactivation of Tuberculosis in Mice Caused byMycobacterium tuberculosisDisrupted inmce1R

Author(s) -

ChanIck Cheigh,

Ryan H. Senaratne,

Yujiro Uchida,

Nicola Casali,

Lon V. Kendall,

Lee W. Riley

Publication year - 2010

Publication title -

the journal of infectious diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.69

H-Index - 252

eISSN - 1537-6613

pISSN - 0022-1899

DOI - 10.1086/655224

Subject(s) - mycobacterium tuberculosis , mutant , tuberculosis , operon , biology , isoniazid , pyrazinamide , microbiology and biotechnology , latent tuberculosis , mutation , gene , virology , immunology , medicine , genetics , pathology

The reactivation of tuberculosis arises in persons who are latently infected and in those who have been previously treated. The mechanism of the reactivation of tuberculosis in either situation is not well understood. A 13-gene mce1 operon of Mycobacterium tuberculosis was previously shown to be associated with latent infection in mice and may also play a role in reactivation.

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