Immunologic and Virologic Events in Early HIV Infection Predict Subsequent Rate of Progression | Zendy

Anuradha Ganesan | Zendy; Pratip K. Chattopadhyay | Zendy; Tess M. Brodie | Zendy; Jing Qin | Zendy; Wenjuan Gu | Zendy; John R. Mascola | Zendy; Nelson L. Michael | Zendy; Dean Follmann | Zendy; Mario Roederer | Zendy

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Immunologic and Virologic Events in Early HIV Infection Predict Subsequent Rate of Progression

Author(s) -

Anuradha Ganesan,

Pratip K. Chattopadhyay,

Tess M. Brodie,

Jing Qin,

Wenjuan Gu,

John R. Mascola,

Nelson L. Michael,

Dean Follmann,

Mario Roederer

Publication year - 2009

Publication title -

the journal of infectious diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.69

H-Index - 252

eISSN - 1537-6613

pISSN - 0022-1899

DOI - 10.1086/649430

Subject(s) - simian immunodeficiency virus , immunology , virology , viral load , disease , pathogenesis , immunopathology , virus , viral disease , human immunodeficiency virus (hiv) , lentivirus , immunodeficiency , biology , medicine , immune system , pathology

Variability in human immunodeficiency virus (HIV) disease progression cannot be fully predicted by CD4(+) T cell counts or viral load (VL). Because central memory T (T(CM)) cells play a critical role in the pathogenesis of simian immunodeficiency virus disease, we hypothesized that quantifying these cells in early HIV infection could provide prognostic information.

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