Relationship between Inflammatory Markers, Endothelial Activation Markers, and Carotid Intima‐Media Thickness in HIV‐Infected Patients Receiving Antiretroviral Therapy | Zendy

Allison C. Ross | Zendy; Nesrine Rizk | Zendy; Mary Ann O’Riordan | Zendy; Vikram S. Dogra | Zendy; Dalia ElBejjani | Zendy; Norma Storer | Zendy; Danielle Harrill | Zendy; Marisa Tungsiripat | Zendy; Jerome Adell | Zendy; Grace A. McComsey | Zendy

Open Access

Relationship between Inflammatory Markers, Endothelial Activation Markers, and Carotid Intima‐Media Thickness in HIV‐Infected Patients Receiving Antiretroviral Therapy

Author(s) -

Allison C. Ross,

Nesrine Rizk,

Mary Ann O’Riordan,

Vikram S. Dogra,

Dalia ElBejjani,

Norma Storer,

Danielle Harrill,

Marisa Tungsiripat,

Jerome Adell,

Grace A. McComsey

Publication year - 2009

Publication title -

clinical infectious diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.44

H-Index - 336

eISSN - 1537-6591

pISSN - 1058-4838

DOI - 10.1086/605578

Subject(s) - medicine , proinflammatory cytokine , endothelial activation , endothelial dysfunction , intima media thickness , c reactive protein , inflammation , surrogate endpoint , intercellular adhesion molecule , immunology , biomarker , gastroenterology , cell adhesion , adhesion , carotid arteries , biochemistry , chemistry , organic chemistry

Human immunodeficiency virus (HIV)-infected patients are at increased risk of cardiovascular disease, which may be related to chronic inflammation and endothelial dysfunction despite virological control with antiretroviral therapy. The relationship between carotid intima-media thickness (IMT), a surrogate marker for cardiovascular disease, proinflammatory cytokines, and endothelial activation markers has not been fully explored in HIV-infected patients who are receiving antiretroviral therapy.

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