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Wound infections caused by Staphylococcus aureus are associated with significant morbidity and mortality. The staphylococcal extracellular adherence protein (Eap) has been shown to delay wound healing by interfering with host defense and angiogenesis, yet the expression of Eap in the human wound is required to exert these functions.

Transcription Analysis of the Extracellular Adherence Protein fromStaphylococcus aureusin Authentic Human Infection and In Vitro