Molecular Epidemiology ofFrancisella tularensisin the United States | Zendy

Kiersten J. Kugeler | Zendy; Paul S. Mead | Zendy; Aimee M. Janusz | Zendy; J. Erin Staples | Zendy; Kristy Kubota | Zendy; Linda Chalcraft | Zendy; Jeannine M. Petersen | Zendy

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Molecular Epidemiology ofFrancisella tularensisin the United States

Author(s) -

Kiersten J. Kugeler,

Paul S. Mead,

Aimee M. Janusz,

J. Erin Staples,

Kristy Kubota,

Linda Chalcraft,

Jeannine M. Petersen

Publication year - 2009

Publication title -

clinical infectious diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.44

H-Index - 336

eISSN - 1537-6591

pISSN - 1058-4838

DOI - 10.1086/597261

Subject(s) - francisella tularensis , tularemia , genotype , biology , molecular epidemiology , typing , virulence , microbiology and biotechnology , subtyping , pulsed field gel electrophoresis , virology , genetics , gene , computer science , programming language

In the United States, tularemia is caused by Francisella tularensis subsps. tularensis (type A) and holarctica (type B). Molecular subtyping has further divided type A into 2 subpopulations, A1 and A2. Significant mortality differences were previously identified between human infections caused by A1 (14%), A2 (0%) and type B (7%). To verify these findings and to further define differences among genotypes, we performed a large-scale molecular epidemiologic analysis of F. tularensis isolates from humans and animals.

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