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Biofilm formation is considered to be an important virulence factor of the opportunistic pathogen Staphylococcus epidermidis. We hypothesized that biofilm formation could interfere with the deposition of immunoglobulins and complement on the bacterial surface, leading to diminished activation of the complement system and protection from killing by human phagocytes.

the journal of infectious diseases (online. university of chicago press)/the journal of infectious diseases

Biofilm Formation Induces C3a Release and Protects<i>Staphylococcus epidermidis</i>from IgG and Complement Deposition and from Neutrophil‐Dependent Killing

Biofilm Formation Induces C3a Release and ProtectsStaphylococcus epidermidisfrom IgG and Complement Deposition and from Neutrophil‐Dependent Killing