Lack of Association between Group B Meningococcal Disease and Autoimmune Disease | Zendy

Michael Frantz Howitz | Zendy; Tyra Grove Krause | Zendy; Jacob Simonsen | Zendy; Steen Hoffmann | Zendy; Morten Frisch | Zendy; Nete Munk Nielsen | Zendy; Jennifer Robbins | Zendy; R. Schneerson | Zendy; Kåre Mølbak | Zendy; Mark A. Miller | Zendy

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Lack of Association between Group B Meningococcal Disease and Autoimmune Disease

Author(s) -

Michael Frantz Howitz,

Tyra Grove Krause,

Jacob Simonsen,

Steen Hoffmann,

Morten Frisch,

Nete Munk Nielsen,

Jennifer Robbins,

R. Schneerson,

Kåre Mølbak,

Mark A. Miller

Publication year - 2007

Publication title -

clinical infectious diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.44

H-Index - 336

eISSN - 1537-6591

pISSN - 1058-4838

DOI - 10.1086/522190

Subject(s) - medicine , meningococcal disease , disease , autoimmune disease , immunology , association (psychology) , group b , neisseria meningitidis , genetics , bacteria , biology , philosophy , epistemology

The capsular polysaccharide of group B meningococci (GBM) is structurally identical to a polysaccharide found on neural cell adhesion molecules in humans. This structural identity has raised concern that a vaccine based on the GBM capsular polysaccharide might induce autoimmune disease in vaccinated persons. Because systemic infection with GBM induces serum antibody in adults, we investigated whether persons with a history of GBM disease are at increased risk of developing autoimmune diseases.

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