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Role of Fibrinogen‐Binding Adhesin Expression in Septic Arthritis and Septicemia Caused byStreptococcus agalactiae
Author(s) -
IngMarie Jonsson,
Giampiero Pietrocola,
Pietro Speziale,
Margareta Verdrengh,
Andrzej Tarkowski
Publication year - 2005
Publication title -
the journal of infectious diseases
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.69
H-Index - 252
eISSN - 1537-6613
pISSN - 0022-1899
DOI - 10.1086/491478
Subject(s) - streptococcus agalactiae , microbiology and biotechnology , bacterial adhesin , virulence , sepsis , septic arthritis , biology , streptococcus , arthritis , immunology , meningitis , fibrinogen , pathogen , medicine , bacteria , gene , biochemistry , genetics , psychiatry
Streptococcus agalactiae (group B streptococcus) is an important human pathogen that causes neonatal pneumonia, sepsis, septic arthritis, and meningitis, as well as severe infections in immunocompromised adult patients. The streptococci produce several molecules important for virulence.

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