Role of Fibrinogen‐Binding Adhesin Expression in Septic Arthritis and Septicemia Caused byStreptococcus agalactiae
Author(s) -
IngMarie Jonsson,
Giampiero Pietrocola,
Pietro Speziale,
Margareta Verdrengh,
Andrzej Tarkowski
Publication year - 2005
Publication title -
the journal of infectious diseases
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.69
H-Index - 252
eISSN - 1537-6613
pISSN - 0022-1899
DOI - 10.1086/491478
Subject(s) - streptococcus agalactiae , microbiology and biotechnology , bacterial adhesin , virulence , sepsis , septic arthritis , biology , streptococcus , arthritis , immunology , meningitis , fibrinogen , pathogen , medicine , bacteria , gene , biochemistry , genetics , psychiatry
Streptococcus agalactiae (group B streptococcus) is an important human pathogen that causes neonatal pneumonia, sepsis, septic arthritis, and meningitis, as well as severe infections in immunocompromised adult patients. The streptococci produce several molecules important for virulence.
Accelerating Research
Robert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom
Address
John Eccles HouseRobert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom