Low Accumulation of L90M in Protease from Subtype F HIV‐1 with Resistance to Protease Inhibitors Is Caused by the L89M Polymorphism | Zendy

Alexandre Calazans | Zendy; Rodrigo Brindeiro | Zendy; Patrícia A. Brindeiro | Zendy; Hugo Verli | Zendy; Mônica Arruda | Zendy; Luis M. F. Gonzalez | Zendy; Jorge A. Guimarães | Zendy; Ricardo Sobhie Diaz | Zendy; O. A. C. Antunes | Zendy; Amílcar Tanuri | Zendy

Open Access

Low Accumulation of L90M in Protease from Subtype F HIV‐1 with Resistance to Protease Inhibitors Is Caused by the L89M Polymorphism

Author(s) -

Alexandre Calazans,

Rodrigo Brindeiro,

Patrícia A. Brindeiro,

Hugo Verli,

Mônica Arruda,

Luis M. F. Gonzalez,

Jorge A. Guimarães,

Ricardo Sobhie Diaz,

O. A. C. Antunes,

Amílcar Tanuri

Publication year - 2005

Publication title -

the journal of infectious diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.69

H-Index - 252

eISSN - 1537-6613

pISSN - 0022-1899

DOI - 10.1086/430002

Subject(s) - biology , clone (java method) , virology , resistance mutation , mutation , protease , virus , viral replication , mutagenesis , genetics , phenotype , mutant , gene , enzyme , reverse transcriptase , polymerase chain reaction , biochemistry

This work evaluates the role of subtype F human immunodeficiency virus type 1 (HIV-1) protease (PR) substitutions L89M and L90M in viral replication and resistance to PR inhibitors (PIs).

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