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This work evaluates the role of subtype F human immunodeficiency virus type 1 (HIV-1) protease (PR) substitutions L89M and L90M in viral replication and resistance to PR inhibitors (PIs).

Low Accumulation of L90M in Protease from Subtype F HIV‐1 with Resistance to Protease Inhibitors Is Caused by the L89M Polymorphism