Exosomes derived from LPS-stimulated human thymic mesenchymal stromal cells enhance inflammation via thrombospondin-1
Author(s) -
Qianru Li,
Jing Li,
Lei Sun,
Yun Sun,
Fei Zhao,
Pingping Liu,
Xin Peng,
Xiaoyan Xuan,
Yun Li,
Peng Wang,
Chen Tan,
Ying Du
Publication year - 2021
Publication title -
bioscience reports
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.938
H-Index - 77
eISSN - 1573-4935
pISSN - 0144-8463
DOI - 10.1042/bsr20203573
Subject(s) - microvesicles , mesenchymal stem cell , stromal cell , inflammation , thrombospondin 1 , microbiology and biotechnology , thrombospondin , biology , immune system , tumor necrosis factor alpha , chemistry , cancer research , immunology , metalloproteinase , microrna , matrix metalloproteinase , angiogenesis , biochemistry , gene
Inflammatory response mediated by immune cells is either directly or indirectly regulated by mesenchymal stromal cells (MSCs). Accumulating evidence suggests that thrombospondin-1 (TSP-1) is highly expressed in response to inflammation. In this work, we isolated and identified human thymic mesenchymal stromal cells (tMSCs) and detected the expression of TSP-1. We found that tMSCs expressed TSP-1 and Poly (I:C) or LPS treatment promoted the expression of TSP-1. Further, we isolated and identified exosomes originating from tMSCs (MEXs). Notably, exosomes derived from LPS-pretreated tMSCs (MEXsLPS) promoted the polarization of macrophages to M1-like phenotype and IL-6, TNF-α secretion as well as the pro-inflammatory differentiation of CD4+T cells into Th17 cells. Upon silencing the expression of TSP-1 in tMSCs, the pro-inflammatory effects of MEXsLPS were suppressed. Therefore, these findings uncovered TSP-1 as the principal factor in MEXsLPS pro-inflammatory regulation.
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