Long non-coding RNA PRNCR1 modulates non-small cell lung cancer cell proliferation, apoptosis, migration, invasion, and EMT through PRNCR1/miR-126-5p/MTDH axis | Zendy

Ran Guo | Zendy; Tongchen Hu | Zendy; Yanheng Liu | Zendy; Yinzai He | Zendy; Yanling Cao | Zendy

Open Access

Long non-coding RNA PRNCR1 modulates non-small cell lung cancer cell proliferation, apoptosis, migration, invasion, and EMT through PRNCR1/miR-126-5p/MTDH axis

Author(s) -

Ran Guo,

Tongchen Hu,

Yanheng Liu,

Yinzai He,

Yanling Cao

Publication year - 2020

Publication title -

bioscience reports

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.938

H-Index - 77

eISSN - 1573-4935

pISSN - 0144-8463

DOI - 10.1042/bsr20193153

Subject(s) - cell growth , cancer research , gene knockdown , flow cytometry , microrna , vimentin , apoptosis , cell , cell migration , biology , long non coding rna , competing endogenous rna , epithelial–mesenchymal transition , cell culture , microbiology and biotechnology , rna , cancer , metastasis , immunology , gene , immunohistochemistry , genetics , biochemistry

Non-small cell lung cancer (NSCLC) is a highly malignant tumor. Accumulating evidence suggested that prostate cancer non-coding RNA 1 (PRNCR1) participated in the pathogenesis of NSCLC, whereas the elaborate mechanism remains unclear. Hence, the role of PRNCR1 in the progression of NSCLC was investigated.

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