Open AccessGestational caffeine exposure acts as a fetal thyroid-cytokine disruptor by activating caspase-3/BAX/Bcl-2/Cox2/NF-κB at ED 20
Author(s) -
R.G. Ahmed
Publication year - 2018
Publication title -
toxicology research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.709
H-Index - 31
eISSN - 2045-4538
pISSN - 2045-452X
DOI - 10.1039/c8tx00227d
Subject(s) - caffeine , cytokine , medicine , apoptosis , caspase 3 , fetus , andrology , pharmacology , endocrinology , chemistry , pregnancy , biology , programmed cell death , biochemistry , genetics
The objective of this examination was to explore the impact of gestational caffeine (1,3,7-trimethylxanthine) exposure on the maternofetal thyroid axis and fetal thyroid–cytokine communications during gestation.
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