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Cigarette smoke compounds induce cellular redox imbalance, activate NF-κB, and increase TNF-α/CRP secretion: a possible pathway in the pathogenesis of COPD
Author(s) -
Tapan Dey,
Prachurjya Dutta,
Prasenjit Manna,
Jatin Kalita,
Hari Prasanna Deka Boruah,
Alak Kumar Buragohain,
Balagopalan Unni,
Dibyajyoti Ozah,
Mihir Kumar Goswami,
Ratan Kumar Kotokey
Publication year - 2016
Publication title -
toxicology research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.709
H-Index - 31
eISSN - 2045-4538
pISSN - 2045-452X
DOI - 10.1039/c5tx00477b
Subject(s) - pathogenesis , copd , cigarette smoke , secretion , tumor necrosis factor alpha , medicine , nf κb , pulmonary disease , inflammation , cigarette smoking , immunology , environmental health
Cigarette smoke has always been considered as a risk factor for chronic obstructive pulmonary diseases (COPD).

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