Low-dose chemotherapeutic drugs induce reactive oxygen species and initiate apoptosis-mediated genomic instability | Zendy

Arun Renganathan | Zendy; Dhivya Sridaran | Zendy; Suresh K. Abraham | Zendy; Kumpati Premkumar | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Low-dose chemotherapeutic drugs induce reactive oxygen species and initiate apoptosis-mediated genomic instability

Author(s) -

Arun Renganathan,

Dhivya Sridaran,

Suresh K. Abraham,

Kumpati Premkumar

Publication year - 2016

Publication title -

toxicology research

Language(s) - Uncategorized

Resource type - Journals

SCImago Journal Rank - 0.709

H-Index - 31

eISSN - 2045-4538

pISSN - 2045-452X

DOI - 10.1039/c5tx00391a

Subject(s) - apoptosis , etoposide , dna damage , cancer cell , genome instability , reactive oxygen species , cell cycle , cancer research , programmed cell death , doxorubicin , cancer , biology , population , cell , pharmacology , chemotherapy , medicine , microbiology and biotechnology , dna , genetics , environmental health

Low-dose chemotherapy drugs initiate defective apoptosis, allowing a small population of cells to escape from cell cycle check points and survive with accumulated genetic damage.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research