Pulmonary Edema in COVID19—A Neural Hypothesis | Zendy

Anoop U.R. | Zendy; Kavita Verma | Zendy

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Pulmonary Edema in COVID19—A Neural Hypothesis

Author(s) -

Anoop U.R.,

Kavita Verma

Publication year - 2020

Publication title -

acs chemical neuroscience

Language(s) - Uncategorized

Resource type - Journals

SCImago Journal Rank - 1.158

H-Index - 69

ISSN - 1948-7193

DOI - 10.1021/acschemneuro.0c00370

Subject(s) - medicine , exacerbation , pathogenesis , pulmonary edema , edema , pneumonia , inflammation , lung , pathology , coronavirus , thrombosis , immunology , covid-19 , disease , infectious disease (medical specialty)

In COVID-19, lung manifestations present as a slowly evolving pneumonia with insidious early onset interstitial pulmonary edema that undergoes acute exacerbation in the late stages and microvascular thrombosis. Currently, these manifestations are considered to be only consequences of pulmonary SARS-CoV-2 virus infection. We are proposing a new hypothesis that neurogenic insult may also play a major role in the pathogenesis of these manifestations. SARS-CoV-2 mediated inflammation of the nucleus tractus solitarius (NTS) may play a role in the acute exacerbation of pulmonary edema and microvascular clotting in COVID-19 patients.

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