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Ischemia-reperfusion injury is a major factor in the early phase of lung transplantation. We hypothesized that aprotinin, a nonspecific serine protease inhibitor, attenuates ischemia-reperfusion lung injury by inhibiting the inflammatory response and suppressing NADPH oxidase.

Aprotinin attenuated ischemia–reperfusion injury in an isolated rat lung model after 18-hours preservation