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ERBB3 overexpression due to miR-205 inactivation confers sensitivity to FGF, metabolic activation, and liability to ERBB3 targeting in glioblastoma
Author(s) -
Francesca De Bacco,
Francesca Orzan,
Jessica Erriquez,
Elena Casanova,
Ludovic Barault,
Raffaella Albano,
Antonio D’Ambrosio,
Viola Bigatto,
Gigliola Reato,
Monica Patanè,
Bianca Pollo,
Geoffrey Kuesters,
Carmine Dell’Aglio,
Laura Casorzo,
Serena Pellegatta,
Gaetano Finocchiaro,
Paolo M. Comoglio,
Carla Boccaccio
Publication year - 2021
Publication title -
cell reports
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 6.264
H-Index - 154
eISSN - 2639-1856
pISSN - 2211-1247
DOI - 10.1016/j.celrep.2021.109455
Subject(s) - erbb3 , cancer research , pi3k/akt/mtor pathway , gene silencing , biology , downregulation and upregulation , protein kinase b , signal transduction , microbiology and biotechnology , receptor tyrosine kinase , gene , genetics

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