Physical interactions between MCM and Rad51 facilitate replication fork lesion bypass and ssDNA gap filling by non-recombinogenic functions | Zendy

María J. Cabello-Lobato | Zendy; Cristina GonzálezGarrido | Zendy; María I. Cano-Linares | Zendy; Ronald P. Wong | Zendy; Aurora YáñezVilches | Zendy; Macarena MorilloHuesca | Zendy; Juan María RoldánRomero | Zendy; Marta Vicioso | Zendy; Román GonzálezPrieto | Zendy; Helle D. Ulrich | Zendy; Félix Prado | Zendy

Open Access

Physical interactions between MCM and Rad51 facilitate replication fork lesion bypass and ssDNA gap filling by non-recombinogenic functions

Author(s) -

María J. Cabello-Lobato,

Cristina GonzálezGarrido,

María I. Cano-Linares,

Ronald P. Wong,

Aurora YáñezVilches,

Macarena MorilloHuesca,

Juan María RoldánRomero,

Marta Vicioso,

Román GonzálezPrieto,

Helle D. Ulrich,

Félix Prado

Publication year - 2021

Publication title -

cell reports

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 6.264

H-Index - 154

eISSN - 2639-1856

pISSN - 2211-1247

DOI - 10.1016/j.celrep.2021.109440

Subject(s) - rad51 , rad52 , minichromosome maintenance , homologous recombination , microbiology and biotechnology , helicase , biology , control of chromosome duplication , dna replication , dna repair , scaffold protein , replication protein a , replication factor c , dna , dna binding protein , genetics , gene , transcription factor , rna , signal transduction

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