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NADK is activated by oncogenic signaling to sustain pancreatic ductal adenocarcinoma
Author(s) -
Tanya Schild,
Melanie R. McReynolds,
Christie Shea,
Vivien Low,
Bethany E. Schaffer,
John M. Asara,
Elena Piskounova,
Noah Dephoure,
Joshua D. Rabinowitz,
Ana P. Gomes,
John Blenis
Publication year - 2021
Publication title -
cell reports
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 6.264
H-Index - 154
eISSN - 2639-1856
pISSN - 2211-1247
DOI - 10.1016/j.celrep.2021.109238
Subject(s) - nad+ kinase , hyperactivation , phosphorylation , kras , pancreatic ductal adenocarcinoma , metabolic adaptation , protein kinase c , kinase , cancer research , enzyme , signal transduction , biology , pancreatic cancer , microbiology and biotechnology , glycolysis , chemistry , biochemistry , cancer , metabolism , gene , mutation , genetics

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