Olmesartan Ameliorates Renovascular Injury and Oxidative Stress in Zucker Obese Rats Enhanced by Dietary Protein | Zendy

Tamehachi Namikoshi | Zendy; Natsuo Tomita | Zendy; Masataka Satoh | Zendy; Yoshimichi Haruna | Zendy; Shotai Kobayashi | Zendy; N KOMAI | Zendy; Takashi Sasaki | Zendy; Naoki Kashihara | Zendy

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Olmesartan Ameliorates Renovascular Injury and Oxidative Stress in Zucker Obese Rats Enhanced by Dietary Protein

Author(s) -

Tamehachi Namikoshi,

Natsuo Tomita,

Masataka Satoh,

Yoshimichi Haruna,

Shotai Kobayashi,

N KOMAI,

Takashi Sasaki,

Naoki Kashihara

Publication year - 2007

Publication title -

american journal of hypertension

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.009

H-Index - 136

eISSN - 1941-7225

pISSN - 0895-7061

DOI - 10.1016/j.amjhyper.2007.05.007

Subject(s) - olmesartan , medicine , endocrinology , hydralazine , p22phox , oxidative stress , angiotensin ii , glomerulosclerosis , metabolic syndrome , endothelial dysfunction , kidney , nadph oxidase , proteinuria , diabetes mellitus , blood pressure

The metabolic syndrome is a risk factor for the development of renal and vascular complications. Dietary protein intake aggravates renal injury in Zucker obese rats, a model of the metabolic syndrome. This study investigated whether dietary protein intake enhances renal and vascular injuries by oxidative stress, and assessed effects of olmesartan, an angiotensin II type 1 receptor blocker, in this model.

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