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Previous studies have demonstrated a high correlation between arterial hypertension and the development of lesions in the carotid glomus (CG) and autonomic ganglia (AG), characterized by extracellular matrix (ECM) expansion and reduction in the number of AG neurons. Because lowering blood pressure (BP) is the first step in controlling the deleterious effects of arterial hypertension, the objective was to evaluate possible differences between the beta-blocker atenolol (AT) and the angiotensin-converting enzyme (ACE) inhibitor ramipril (RAM) regarding a protective role on CG and AG, as target organs in the spontaneously hypertensive rat (SHR).

american journal of hypertension

Effects of ACE Inhibition and β-Blockade on Plasminogen Activator Inhibitor-1 and Transforming Growth Factor-β1 in Carotid Glomus and Autonomic Ganglia in Hypertensive Rats