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Cerebral edema is an important initial event in cases of stroke among humans. Although hypertension is a major risk factor for endothelial injury, the precise mechanisms regulating brain microvascular changes are still unknown. To elucidate the pathogenesis of increases in vascular permeability in the cerebral cortex, we investigated the expression of glucose transporter-1 (GLUT-1) in endothelial cells and aquaporin-4 (AQP4) in astrocytes in relation to blood-brain barrier (BBB) function.

american journal of hypertension

Expression of Glucose Transporter-1 and Aquaporin-4 in the Cerebral Cortex of Stroke-Prone Spontaneously Hypertensive Rats in Relation to the Blood-Brain Barrier Function