IL-22/IL-22R1 signaling regulates the pathophysiology of chronic rhinosinusitis with nasal polyps via alteration of MUC1 expression | Zendy

Yasuyuki Noyama | Zendy; Mitsuhiro Okano | Zendy; Tazuko Fujiwara | Zendy; Shin Kariya | Zendy; Takaya Higaki | Zendy; Takenori Haruna | Zendy; Seiichiro Makihara | Zendy; Kengo Kanai | Zendy; Takahisa Koyama | Zendy; Masami Taniguchi | Zendy; Jun-ichi Ishitoya | Zendy; Akira Kanda | Zendy; Yoshiki Kobayashi | Zendy; Mikiya Asako | Zendy; Koichi Tomoda | Zendy; Kazunori Nishizaki | Zendy

Open Access

IL-22/IL-22R1 signaling regulates the pathophysiology of chronic rhinosinusitis with nasal polyps via alteration of MUC1 expression

Author(s) -

Yasuyuki Noyama,

Mitsuhiro Okano,

Tazuko Fujiwara,

Shin Kariya,

Takaya Higaki,

Takenori Haruna,

Seiichiro Makihara,

Kengo Kanai,

Takahisa Koyama,

Masami Taniguchi,

Jun-ichi Ishitoya,

Akira Kanda,

Yoshiki Kobayashi,

Mikiya Asako,

Koichi Tomoda,

Kazunori Nishizaki

Publication year - 2016

Publication title -

allergology international

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.49

H-Index - 58

eISSN - 1440-1592

pISSN - 1323-8930

DOI - 10.1016/j.alit.2016.04.017

Subject(s) - nasal polyps , cytokine , medicine , pathogenesis , inflammation , eosinophilia , immunology , pathology

IL-22 is an IL-10-family cytokine that regulates chronic inflammation. We investigated the role of IL-22 and its receptor, IL-22R1, in the pathophysiology of chronic rhinosinusitis with nasal polyps (CRSwNP).

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